Comparison of host genetic factors influencing pig response to infection with two North American isolates of porcine reproductive and respiratory syndrome virus

Table 5 Estimates of correlations^a (standard error) of response to infection with PRRSV isolate NVSL

Trait	VL	WG	TP	PV	Tmax	Vmax
VL		−0.33 (0.03)	0.10 (0.03)	0.66 (0.02)	0.36 (0.03)	−0.27 (0.03)
WG	−0.74 (0.10)		−0.02 (0.03)	−0.22 (0.03)	−0.16 (0.03)	0.12 (0.03)
TP	0.31 (0.15)	0.27 (0.16)		−0.09 (0.03)	0.72 (0.01)	0.12 (0.03)
PV	0.85 (0.07)	−0.73 (0.13)	0.05 (0.19)		−0.23 (0.03)	0.40 (0.03)
Tmax	0.81 (0.10)	−0.11 (0.16)	0.83 (0.07)	0.50 (0.21)		−0.51 (0.02)
Vmax	−0.72 (0.21)	0.45 (0.22)	−0.11 (0.26)	−0.27 (0.33)	−0.57 (0.19)

WG weight gain (kg), VL viral load (area under the Wood’s curve of log₁₀ serum viremia from 0 to 21 days post infection; viremia * days), TP time to peak viremia (days), PV peak viremia (log₁₀ serum viremia), Tmax time to maximal rate of viremia decay (days), Vmax maximal rate of viremia decay (log₁₀ serum viremia/day)
^aPhenotypic correlations (above diagonal) and genetic correlations (below diagonal) were estimated using an animal model in ASReml and the full G-matrix

ISSN: 1297-9686