Comparison of host genetic factors influencing pig response to infection with two North American isolates of porcine reproductive and respiratory syndrome virus

Table 6 Correlations of response^a to infection with PRRSV isolate KS06 using the full G-matrix

Trait	VL	WG	TP	PV	Tmax	Vmax
VL		−0.23 (0.05)	−0.06 (0.05)	0.76 (0.02)	0.13 (0.05)	−0.16 (0.05)
WG	−0.52 (0.17)		−0.05 (0.05)	−0.13 (0.05)	−0.06 (0.06)	0.13 (0.05)
TP	−0.08 (0.22)	−0.10 (0.24)		0.02 (0.05)	0.80 (0.02)	0.02 (0.05)
PV	0.91 (0.05)	−0.30 (0.18)	−0.08 (0.25)		−0.19 (0.05)	0.52 (0.04)
Tmax	0.19 (0.23)	−0.42 (0.23)	0.69 (0.19)	−0.24 (0.28)		−0.52 (0.04)
Vmax	−0.01 (0.20)	0.42 (0.21)	−0.12 (0.28)	0.51 (0.13)	−0.75 (0.18)

WG weight gain (kg), VL viral load (area under the Wood’s curve of log₁₀ serum viremia from 0 to 21 days post infection; viremia * days), TP time to peak viremia (days), PV peak viremia (log₁₀ serum viremia), Tmax time to maximal rate of viremia decay (days), Vmax maximal rate of viremia decay (log₁₀ serum viremia/day)
^aPhenotypic correlations (above diagonal) and genetic correlations (below diagonal) were estimated using an animal model in ASReml and the full G-matrix

ISSN: 1297-9686